At any rate,it is documented to increase appetite so using it for weight loss kind of sucks in that regard.

So Clen,isn't for far loss..?

Doesnt it lower mTOR?

In summary, the present findings demonstrate that the 2-adrenergic agonist clenbuterol does activate Akt and its downstream effector mTOR. Activation of mTOR and its downstream targets 4E-BP1 and S6K1 can lead to increases in protein translation and could explain previous findings of increased protein synthesis following clenbuterol treatment. The ability of clenbuterol to repress MuRF1 and MAFbx transcripts and components of the ubiquitin proteosome pathways (48) suggests that clenbuterol can also affect protein degradation. The findings suggest that activation of mTOR is critical for clenbuterol-induced muscle growth in normal animals and muscle sparing during unloading. The mechanism through which clenbuterol activates mTOR is likely through the activation of Akt, although other mechanisms cannot be ruled out. We postulate that Akt is activated through Gi-G-PI3-kinase signaling pathways (see Fig. 8), similar to what occurs in cardiac tissue. Additional experiments are required to determine whether PI3-kinase is activated by clenbuterol and responsible for the activation of Akt. Further analysis is also needed to parse out the roles of the Akt/mTOR and Gs-AC-cAMP pathways in mediating the actions of clenbuterol in skeletal muscle. It is clear from the data that multiple pathways mediate the effects of clenbuterol in skeletal muscle. A better understanding of the actions of 2-adrenergic receptor agonists in skeletal muscle could lead to the development of compounds to treat muscle atrophy.